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Furthermore, the downregulation of DMT1 enhanced the expression of Nrf2, HO-1, and GPX4

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found that quercetin indirectly prevents inflammation by enhancing peroxisome proliferator-activated receptor γ activity, thereby opposing NF-κB or AP-1 mediated transcription of inflammatory genes, collectively obstructing the TNF-α-mediated inflammatory cascade and reducing inflammation damage from radiation therapy 38